Friday , November 27 2020

Mystery of ‘happy oxygen deficiency’ caused by Covid-19 solutions



How Covid-19 causes oxygen deficiency, also known as silent or happy hypoxia, is one of the biggest and most dangerous mysteries of the disease.

However, the researchers noted that the body’s oxygen level is unusually low and they have begun to lift the throat over this disorder that will irreversibly damage vital organs if left unchecked for a long time.

Scientists from Boston University and Vermont have begun to discover how the coronavirus attacks the lungs and other body parts by comparing data from real patients and modeling on computers, Independent Turkish quoted Medical News.

In the lungs of a healthy person, keeping the oxygen levels in the blood from 95 percent to 100. If it drops below 92 percent, even medical attention will be needed.

However, previous studies have revealed that many Covid-19 patients do not show signs of shortness of breath, even though they have dangerously low oxygen levels.

In these patients, the infection is thought to damage the lungs first, causing some to not function properly. These tissues lose oxygen and stop functioning. Therefore, oxygen deficiency occurs.

However, exactly how this “domino effect” occurs is unknown, while Professor Bela Suki of Boston University, co-author of the study in question, said it was unknown whether the situation was physiologically possible .

Professor Suki also noted that despite this low blood oxygen level, most of these patients showed no abnormalities or very mild symptoms in their lung scans.

Biomedical Engineer Jacob Herrman, lead author of the study published in Nature Communications, noted that a combination of biological mechanisms likely to develop simultaneously in the lungs of Kovid-19 patients causes oxygen deficiency.

A combination of three reasons

In this study, experts identified three different scenarios and tested them on the computer to explain how and why the lungs stop providing oxygen in Covid-19 patients.

We first looked at the ability of the lungs to regulate where the blood goes, and how Covid-19 affected that ability.

Under normal circumstances, if the lungs do not collect too much oxygen due to infection damage, blood vessels narrow in this area, which is a good thing. Because at the narrowing of the veins, oxygen-rich blood is forced to flow, so oxygen can be delivered to the rest of the body.

However, according to Herrmann, there is some Covidien-19 lungs from patients’ blood flow. Losing his ability to limit himself to the damaged area, he probably opened up more veins. This is difficult to detect or measure on a CT scan.

The team then examined, when clotting, how it can affect blood flow in different parts of the lungs.

Experts said that when the lining of the blood vessels becomes inflamed by Covid-19 infection, clots that are too small to be seen on medical scans can form inside the lungs.

Although it was said that this could cause oxygen deficiency, it was also emphasized that it could not cause oxygen deficiency as low as the levels seen in patients.

Finally, the researchers looked at whether Covid-19 interferes with the air-to-blood flow rate that the lungs need to operate at a standard level.

Incompatible air-to-blood flow rate is a condition that already occurs in many respiratory diseases such as asthma.

For this to cause oxygen deficiency, the study found that there must be mismatches in parts of the lung that do not appear to be injured or abnormal on scans.

The findings suggested that a combination of these three cases was responsible for oxygen deficiency in Covid-19 patients.

Professor Suki also said that each patient responds differently to the virus and that understanding the low oxygen in the blood is important in developing appropriate treatment for the patient.


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